The Driscoll Theory

The Role of External Communicating Hydrocephalus, Mast Cell Disease and CCSVI as the cause of Postural Orthostatic Tachycardia Syndrome in Ehlers-Danlos Syndrome The author explains how pilot studies indicate that External Communicating Hydrocephalus appears to be responsible for the autonomic symptoms (“POTS” – Postural Orthostatic Tachycardia Syndrome) found in 50-70% (or more) of the Classic, Hypermobile and Vascular Ehlers-Danlos Syndrome patient populations....
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The Role of External Communicating Hydrocephalus, Mast Cell Disease and CCSVI as the cause of Postural Orthostatic Tachycardia Syndrome in Ehlers-Danlos Syndrome The author explains how pilot studies indicate that External Communicating Hydrocephalus appears to be responsible for the autonomic symptoms (“POTS” – Postural Orthostatic Tachycardia Syndrome) found in 50-70% (or more) of the Classic, Hypermobile and Vascular Ehlers-Danlos Syndrome patient populations. Retrospective examination of head circumference growth in babies who were later diagnosed with EDS (Ehlers-Danlos Syndrome) and POTS, indicates such hydrocephalus is evident. Furthermore, External Communicating Hydrocephalus, often in conjunction with CCSVI (chronic cerebrospinal venous insufficiency), is hypothesized as the cause of the constellation of cranial nerve signs and symptoms, endocrine abnormalities, personality changes, cognitive decline, extreme fatigue, left ventricular diastolic dysfunction and brain atrophy in the Ehlers-Danlos Syndrome patient population. Finally, Mastocytosis (or mast cell activation syndrome) appears to be a common condition found in patients with EDS and POTS, potentially exacerbating brain fog, dementia, personality changes, and fatigue. Although it is presumed that mast cell disease is secondary to POTS, the potential for congenital mast cell disease causing POTS and hydrocephalus cannot yet be dismissed.